Sunday, 4 September 2011

Circovirus in U.S. Swine


This report goes a long way towards explaining why the former President of the OIE criticised Britain in 2001 for failing to make a circovirus (PMWS) epidemic notifiable. (details recorded on the British newsgroup uk.business.agriculture, fully searchable by Google Groups)

Circovirus is nasty. They thought it then and they were right.

He must have known that Britain's devious government veterinarians were too busy covering up to think beyond blaming the innocent for the Classical Swine Fever and, Foot and Mouth epidemics the following year. We knew, and the writer gave evidence to both the Westminster Parliament and OLAF, the serious Fraud Squad of the European Union, accordingly.

Circovirus seems to have arrived in Britain in 1999, probably from Canada. The point of first infection seems to be variously stated as Northern Ireland, Cornwall or East Anglia. It probably does not matter, it spread quickly.

Anyway, the Americans  are now on the case.

Modern testing on old samples will trace the path of transmission.

Full Report here


A   R E T R O S P E C T I V E  S E R O L O G I C A L  SU R V E Y  O F  P C V 2  
EX P O S U R E  I N  U . S .   F E R A L  SW I N E



29 August 2011


Nearly all domestic swine herds are infected with Porcine Circovirus 2  (PCV2) worldwide.  Although infection with the virus does not always  cause disease, a suite of porcine circovirus associated syndromes and diseases (e.g., postweaning multisystemic wasting syndrome) may  occur in domestic swine when coinfections with PCV2 exist or various environmental (husbandry) or  genetic (sow) risk factors are present.  


Vertical and horizontal transmission of PCV2 have been documented, and fecal/oral transmission  has been identified as the most important route of infection within domestic swine facilities.  Limited geographic   sampling of feral swine in  the U.S. has shown that PCV2 exposure in feral swine may mirror exposure levels seen in domestic swine herds,  although many questions remain unanswered.  In some areas, the relatively high apparent prevalence of PCV2 antibodies in feral swine is suggestive of efficient  transmission of the virus within populations, or among feral and domestic populations.


In an effort to further elucidate the geographic distribution of PCV2 in  feral swine and its association with domestic swine production,  the NWDP initiated a retrospective survey of feral  swine populations  from 72 counties in 18 states representing a  range of domestic swine  production levels.  Over 2495 samples  from the NWDP's feral swine  serum archive are being tested  by the Rollins Animal Disease Diagnostic  Laboratory in North  Carolina for antibodies to  PCV2 .  


Preliminary results suggest that statewide PCV2 prevalence in feral swine  may exceed  50% and that the majority of the  states contain PCV2 infected  feral swine.  Once testing is complete, the NWDP also will  evaluate if PCV2  exposure is  correlated with exposure to other endemic swine diseases.  If so,  exposure to PCV2 in feral swine  may assist in evaluating  risk of  disease  transmission to transitional or domestic swine herds.  


For additional information on  this survey when it becomes  available, please contact Brandon Schmit.